Camelina Omega3 – Lipid metabolism (triglycerides)

Camelina Omega3™ is a natural remedy for the lipid metabolism (triglycerides). It is a tonic and a support for the organism.
A natural supplement made of pure Camelina sativa oil, cold pressed without solvents, it is naturally rich in polyunsaturated fatty acids and Omega 3.

– it improves lipid metabolism (triglycerides)
– it contributes to the maintenance of normal cholesterol levels.
– it contrasts the skin dryness.
– it contrasts mucous membrane dryness.
– it has a tonic and a supportive action.
– it contrasts fatigue and tiredness.
– it acts as anti-inflammatory for joints.

Camelina sativa is an oilseed known since the bronze age, that is a member of the Brassicacee family, like Colza, Mustard and Turnip. It is also called false Flax or wild Flax because of its similarity to Flax oil.
From harvest, an oily seed is obtained, from which come an oil rich in Omega 3, Omega 6 and other polyunsaturated fatty acids. It has a perfect balance between Omega-3 and Omega-6 (2:1 ratio). It is also an excellent source of  Vitamin E, known fot its anti-oxidant properties.

Camelina’s crop has a very low environmental impact thanks to its adaptability to low temperatures and semi-arid climates. It could be cultivated also in marginal areas. It is a very strong plant that can resist to several disease, parasites and insects; for this reason, it doesn’t need plant protection products or herbicides.
For these reasons Camelina’s crop is useful to development in poor countries and areas and doesn’t create pollution or over-exploitation of the soil.

Bibliography:
1. Nì Eidhin D, Burke J, O’Beirne D (2003). Oxidative stability of ω3-rich Camelina oil and Camelina oil-based spread compared with plant and fish oils and sunflower spread. Journal of food science, vol. 68, nr. 1, pp; 345-353.
2. Karvonen M H, Aro A, Tapola S N, Salminen I, Uusitupa M I J, Sarkkinen E S ( 2002). Effect of α-Linolenic acid-rich Camelina sativa oil on serum fatty acid composition and serum lipids in hypercholesterolemic subjects. Metabolism, vol. 51, nr. 10; pp. 1253-1260.
3. Ren J, Chung S H ( 2007). Anti-inflammatory effect of α-Linolenic acid and its mode of action through the inhibition of nitric oxide production and inducible nitric oxide synthase gene expression via NF-κB and mitogen-activated protein kinase pathways. J. Agric. Food Chem., vol. 55, nr. 13; pp. 5073-5080.
4. Mozaffarian D (2005). Does alpha-linolenic acid intake reduce the risk of coronary heart disease? A review of the evidence. Alternative therapies, vol. 11, nr. 3; pp. 24-30